“Accidents are the most wonderful things in this world.

They are wonderfully indescribable, unpredictable, and elusive.

Medical accidents, traffic accidents, sudden illnesses, food poisoning, falling objects from high altitudes, falling from high altitudes, fires, drowning, electric shocks...

Human life is so fragile.

A glass of water, an icicle, a chopstick, a fork, or even a nut is enough to kill someone.

In modern life, people face too many tools every day. The possible ways to die are even more bizarre and flashy than those shown in the movie *Final Destination* or the TV show *1000 Ways to Die*.”

The boy spread his hands and smiled. “Death is everyone's final destination and the fairest thing in the world.

If murder is shocking and infuriating, it will be recorded and countless people will work together to investigate the case and the truth behind it.

Then accidental death has a tone of sorrow, helplessness, and sometimes a bit of black humor.

Similarly, no one cares much about the opposite of accidental death, not even the media chasing traffic—it's an accident, an unavoidable fate.

And I am responsible for creating accidents.”

Perhaps this statement sounded a bit boastful, as the boy’s smile was slightly shy. “As time goes on, I become more and more proficient in this area.

I can even show the entire accident process to the world, and no one can find the complete story before it ends.

Just like a world-class magician, or a cold-blooded and ruthless criminal conspiracy scholar in a detective novel.”

——————

“Sister Lan, do you suspect someone is hunting down these criminals?”

“Yes.”

Wei Linglan nodded, looking at Chu Yin, who was flipping through the documents, and nodded. “These cases are all ordinary, but I always feel like something is not quite right.

I'm not an expert in this area, and my score in the bureau's criminal investigation course wasn't very high.

You're smarter, see if there are any problems here.”

Chu Yin hummed in agreement, took a sip of the nearly empty Coke with ice, and stared intently at the documents.

“Huang Yuanwei, male, 46 years old, driver, previously a contact person in a criminal gang.

One morning in the summer of the year before last, while driving to a neighboring city, he suddenly felt his breathing becoming more and more difficult, his throat and chest tightening.

Needle-like pains kept coming from his fingertips, his hands could barely grip the steering wheel, and the road ahead began to double.

For safety's sake, he barely managed to drive off the highway and immediately went to the hospital for treatment.

When he arrived at the emergency department, his condition was already very bad. His face was morbidly red, he was gasping for breath, and his chest was rising and falling sharply, but the physical and neurological examination results were relatively normal.

Considering that he had eaten local food while on a business trip out of town yesterday, combined with his previous history of allergies,

the doctor thought it was a common allergic reaction and gave him the antihistamine diphenhydramine—this drug has the effect of antagonizing histamine h1 receptors, can inhibit the central nervous system, and is suitable for allergic diseases of the skin and mucous membranes, such as urticaria, hay fever, allergic rhinitis, etc.

Histamine is an active amine compound that exists in mast cells, and also in the mucous membrane tissues of the lungs, liver, and stomach.

When the body detects an allergen, it releases histamine,

and histamine, as a chemical transmitter, binds to receptors on the smooth muscle of blood vessels, causing vasodilation and thus local edema,

and causing the smooth muscle of the trachea in the lungs to contract, causing airway narrowing and thus breathing difficulties,

lowering blood pressure and accelerating the heart rate.

The doctor used diphenhydramine to block the histamine, and gave Huang Yuanwei epinephrine to constrict his dilated blood vessels.

If it was an allergic reaction, then this should work.

But after several hours of observation, Huang Yuanwei's condition did not improve. He still felt uncomfortable and claimed that his stomach was burning, even after drinking an oral antacid.

Then, he felt his throat tighten and swell, unable to speak or swallow, his heart rate rapidly increased, and his eyelids drooped.

The doctor immediately performed various examinations, including MRI, but the examination results did not show any problems.

Huang Yuanwei's condition continued to worsen at an almost bizarre rate.

His facial and throat muscles were paralyzed, he could not speak, his hands and feet could not move, his eyelids drooped and he could not open them, and he could only breathe with the help of a respirator.

The most terrifying thing was that his will was still clear.

He could clearly feel himself losing control of all his muscles bit by bit.

He could clearly feel his body constantly being paralyzed.

Before the doctor could carry out further treatment, he lost his life.

It was not until after his death that the doctors understood the cause of his death.

His acute muscle paralysis was not a problem of the brain or spine, because his systemic paralysis was very balanced, average, and synchronized.

Nor could it be a viral infection of the nerves—viruses such as polio, which can cause systemic paralysis, would cause the patient to have a fever for several days before causing muscle paralysis.

Therefore, it could only be a problem with the neuromuscular junction.

Motor neurons located in the anterior horn of the spinal cord and some nerve nuclei in the brainstem send out nerve fibers to the muscles. At the muscle fibers, the nerve fibers each branch into dozens or hundreds of branches.

Each branch terminates on the muscle fiber, forming a one-to-one neuromuscular junction.

The neuromuscular junction can transmit the signal of the nerve fiber to the muscle fiber, causing the muscle to contract. If the neuromuscular junction is interrupted, it will lead to muscle atrophy and paralysis.”

Chu Yin frowned and read, “And what causes the neuromuscular junction to fail is...”

“Botulinum toxin.”

The boy smiled and said to the old woman, “You see, the entire process of muscle movement

is initiated by the nerve center sending out instructions,

allowing nerve impulses to be transmitted to the axon terminal,

stimulating the presynaptic vesicles to release neurotransmitters such as acetylcholine, which diffuse into the synaptic cleft, bind to receptors on the muscle membrane, and trigger muscle contraction.

Botulinum toxin binds to the presynaptic membrane of cholinergic neurons, undergoes endocytosis, and forms an acidic vesicle containing toxin molecules, which remains at the end of the presynaptic membrane of motor neurons,

preventing the release of the normal neurotransmitter acetylcholine,

thereby paralyzing the muscle.

I have been observing in secret, and sure enough, afterwards the official, as I expected,

followed the botulinum toxin ingested by Huang Yuanwei and found the food he ate at a restaurant yesterday—a ham that was originally in a can.

And the owner of that restaurant happened to be a murder suspect who had been on the run for many years.

For botulinum toxin to be produced in canned food, the temperature and pH of the external environment must be completely suitable.

This is a perfect and regrettable accident.”